A chronic subdural hematoma is a slow, quiet collection of blood that builds between the brain's surface and its outer covering over the course of weeks or months often starting with a fall or bump too minor to remember. Because it builds so gradually, it can be mistaken for a stroke, dementia, or ordinary aging. Treatments range from observation to surgery to endovascular embolization.
A slow bleed between the brain and its outer covering that builds up over weeks or months.
A subdural hematoma is a collection of blood over the surface of the brain (not within the brain). A chronic subdural hematoma forms when the rate of reabsorption doesn't keep up with the rate of blood leakage, so there can be a gradual build up of fluid and pressure on the brain over weeks to months.
A fall or bump, even one minor enough to be forgotten entirely, can tear a brain surface vein and cause it to leak. Unlike an acute subdural hematoma, a large, sudden clot that follows a severe head injury and is usually a same-day emergency, a chronic one accumulates slowly, and may cause gradual symptoms such as headache or imbalance.
Not every chronic subdural hematoma needs treatment right away. A small, mild collection is often simply watched with a follow-up scan. When treatment is needed, it may involve neurosurgery, such as burr hole drainage or a craniotomy, an endovascular procedure called middle meningeal artery embolization, or a combination of the two, depending on the size and internal makeup of the blood collection.
Use the controls below to grow and age a subdural collection. Observe how the brain shifts in response to a growing collection, and how the internal structure changes the longer the subdural hematoma persists.
Because the bleed builds slowly, its symptoms often look like something else entirely. Select an item to learn more.
A chronic subdural hematoma can occasionally bleed acutely on top of its slow baseline. Any sudden, severe headache, new one-sided weakness, difficulty waking up, or loss of consciousness after a fall or head injury, at any age, needs immediate emergency evaluation. Do not wait to see if it improves.
In many cases, the story starts with a set of small, stretched veins on the surface of the brain.
The brain sits inside the skull wrapped in the dura mater, a tough, fibrous membrane. Between the brain's own surface and the dura is a thin, fluid-filled space, and running across that gap are a handful of small cortical veins, "bridging veins," that carry blood from the brain's surface into the large draining sinuses built into the dura itself.
As the brain gradually shrinks with age — a normal process that speeds up with conditions like chronic alcohol use — the space the veins must cross widens. The bridging veins stretch further to make the same connection, leaving them thinner, tauter, and more fragile than they were years earlier.
In many, though not all, cases, that's where a chronic subdural hematoma begins: a jolt or a fall, often minor enough to be forgotten by the time symptoms appear weeks or even months later, shears one of these stretched veins right where it crosses into the sinus. Because bridging veins carry low-pressure venous blood rather than the higher-pressure blood of an artery, the resulting leak is slow, which is why the blood collects gradually rather than causing an immediate, obvious neurologic change.
Bridging vein shearing is the most common cause, especially in older adults and people taking blood thinners, but it isn't the only one. A subdural hematoma can also form from direct injury to a small artery or vein on the brain's surface, from an underlying bleeding disorder, from the slow enlargement of an earlier acute bleed or, occasionally, with no clear preceding injury at all.
There is rarely one single right answer. Several factors together guide whether we recommend observation, drainage, embolization, or a combination. Select a factor to learn how it weighs in.
These three approaches aren't strictly either/or, and which one (or which combination) is right depends heavily on what the collection looks like on imaging: whether it's a single pocket of fluid, divided by membranes, or partly solid. Many patients, especially with a larger, recurrent, or more complex collection, are offered more than one.
| Burr Hole Drainage | Craniotomy | Middle Meningeal Artery Embolization | |
|---|---|---|---|
| What happens | One or two small, nickel-sized holes are drilled through the skull directly over the collection, and the old blood is drained through a thin catheter. | A larger, though still limited and temporary, section of skull is opened so the surgeon can see directly into the space, open any internal pseudomembranes, and remove both liquid and more solid, subacute clot. | A thin catheter is guided from an artery in the wrist or groin up to the middle meningeal artery, the vessel feeding the abnormal membrane around the collection, which is then intentionally blocked from the inside. |
| Access | Through the skull, directly over the collection | Through the skull, using a larger temporary bone opening that is replaced at the end of surgery | Entirely through the blood vessels; no skull opening at all |
| What it treats | Removes the fluid that has already collected, relieving pressure right away | Removes fluid, internal membranes, and solid or subacute clot that a burr hole and catheter cannot adequately reach | Does not remove existing fluid on its own; it starves the membrane's abnormal blood supply so the collection is less likely to keep growing or return |
| Typical hospital stay | 1–3 days, with a drain often left in for 24–48 hours | Typically 3–5 days, reflecting the larger opening and more extensive procedure | 1 additional day if combined with surgery, or a short stay if used alone |
After either burr hole drainage or craniotomy, a thin, soft subdural drain is often left in the space where the collection was for up to a few days afterward. Removing the fluid doesn't immediately close the space it occupied, and that space can slowly refill before the brain has had a chance to re-expand and settle against the skull. A temporary drain lets any fluid that continues to seep in keep draining externally during this early window instead of building back up, which meaningfully lowers the chance of recurrence. It's typically removed at the bedside once imaging or drainage output shows it's no longer needed, before you go home.
From diagnosis to follow-up, the path can branch depending on what's right for you. Select any step below to see what happens and why.
Evidence from clinical series and randomized trials, presented honestly, including the risks.
The most common complication is recurrence, fluid building back up and requiring a repeat procedure, which happens in roughly 10–20% of patients after drainage alone. Seizures can occur in the days to weeks after surgery. Bleeding or infection at the drainage site is uncommon but possible. Craniotomy involves a larger opening than burr hole drainage and carries a modestly higher risk of bleeding or infection, though still low; it is generally reserved for the more complex collections described above, where a burr hole would not adequately drain the membranes or solid clot present. Anesthesia carries its own risks, which are magnified in frail, older patients, and are weighed carefully when planning surgery for this group.
For embolization specifically, risks relate mainly to the catheter access site (bruising, rarely a vessel injury) and, uncommonly, unintended blockage of a nearby vessel. Pooled trial data put the overall complication rate from the embolization procedure itself at around 1%.
Chronic subdural hematoma is managed within Brown Neurosurgery's Neurotrauma division, in partnership with our Endovascular Neurosurgery Program for cases suited to embolization.